Education

Medical School

American University of Beirut
Beirut Lebanon

Internship

American University of Beirut
Beirut Lebanon

Residency

American University of Beirut
Beirut Lebanon

Residency

Boston University, Boston City Hospital
Boston MA

Fellowship

Tufts University, New England Medical Center
Boston MA

Publications

  1. Histological esophagitis: clinical and histological response to omeprazole in children. Dig Dis Sci. 1999 Jan; 44(1):134-9. View Abstract
  2. Developmental delay and growth failure caused by a peroxisomal disorder, dihydroxyacetonephosphate acyltransferase (DHAP-AT) deficiency. Am J Med Genet. 1998 Nov 16; 80(3):223-6. View Abstract
  3. Proarrhythmia associated with cisapride in children. Pediatrics. 1998 Jun; 101(6):1053-6. View Abstract
  4. Maturational regulation of globotriaosylceramide, the Shiga-like toxin 1 receptor, in cultured human gut epithelial cells. J Clin Invest. 1995 Sep; 96(3):1328-35. View Abstract
  5. Developmentally regulated Gb3 galactosyltransferase and alpha-galactosidase determine Shiga toxin receptors in intestine. Am J Physiol. 1994 Oct; 267(4 Pt 1):G618-24. View Abstract
  6. Pathogenesis of Shigella diarrhea: XVII. A mammalian cell membrane glycolipid, Gb3, is required but not sufficient to confer sensitivity to Shiga toxin. J Infect Dis. 1994 Mar; 169(3):538-46. View Abstract
  7. A quantitative immunostaining method for the measurement of UDP-galactose:lactosylceramide galactosyltransferase for the synthesis of globotriaosylceramide in rabbit small intestine and HeLa cells. Anal Biochem. 1993 Oct; 214(1):295-300. View Abstract
  8. Efficacy of cimetidine for gastric acid suppression in pediatric patients. J Pediatr. 1992 Mar; 120(3):474-8. View Abstract
  9. Shiga toxin: intestinal cell receptors and pathophysiology of enterotoxic effects. Rev Infect Dis. 1991 Mar-Apr; 13 Suppl 4:S304-10. View Abstract
  10. Quantitation of the rabbit intestinal glycolipid receptor for Shiga toxin. Further evidence for the developmental regulation of globotriaosylceramide in microvillus membranes. Gastroenterology. 1989 Aug; 97(2):384-91. View Abstract
  11. Pathogenesis of shigella diarrhea: evidence for a developmentally regulated glycolipid receptor for shigella toxin involved in the fluid secretory response of rabbit small intestine. J Infect Dis. 1988 May; 157(5):1023-31. View Abstract
  12. Pathogenesis of Shigella diarrhea: rabbit intestinal cell microvillus membrane binding site for Shigella toxin. Infect Immun. 1986 Aug; 53(2):372-7. View Abstract
  13. Development of carbohydrate absorption in the fetus and neonate. Pediatrics. 1985 Jan; 75(1 Pt 2):160-6. View Abstract

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